Epsom salt

Epsom salt is essentially magnesium sulfate, which can be very beneficial to your body. You need magnesium to help ensure you have proper muscle and nerve growth, as well as enzyme function.

Though little research has been done on the effectiveness of magnesium sulfate on helping with the symptoms of diabetes, there may be some link to it helping with some of its side effects.

Continue reading “Epsom salt”

Helicobacter pylori

Helicobacter pylori (/ˌhɛlɪkɵˈbæktər pˈlɔər/), previously named Campylobacter pylori, is a Gram-negative, microaerophilicbacterium found in the stomach, and may be present in other parts of the body, such as the eye.[1][2][3] It was identified in 1982 by Australian scientists Barry Marshall and Robin Warren with further research led by British scientist Stewart Goodwin, who found that it was present in patients with chronic gastritis and gastric ulcers, conditions not previously believed to have a microbial cause. It is also linked to the development of duodenal ulcers and stomach cancer. However, over 80% of individuals infected with the bacterium are asymptomatic and it may play an important role in the natural stomach ecology.[4]

More than 50% of the world’s population harbor H. pylori in their upper gastrointestinal tract. Infection is more prevalent in developing countries, and incidence is decreasing in Western countries. H. pylori’s helical shape (from which the generic name is derived) is thought to have evolved to penetrate the mucoid lining of the stomach.[5][6]

The pancreas

The pancreas /ˈpæŋkriəs/ is a glandular organ in the digestive system and endocrine system of vertebrates. In humans, it is located in the abdominal cavity behind the stomach. It is an endocrine gland producing several important hormones, including insulin,glucagon, somatostatin, and pancreatic polypeptide which circulate in the blood. The pancreas is also a digestive organ, secretingpancreatic juice containing digestive enzymes that assist digestion and absorption of nutrients in the small intestine. These enzymeshelp to further break down the carbohydrates, proteins, and lipids in the chyme.

The duodenum

The duodenum /ˌdəˈdinəm/ is the first section of the small intestine in most higher vertebrates, including mammals, reptiles, andbirds. In fish, the divisions of the small intestine are not as clear, and the terms anterior intestine or proximal intestine may be used instead of duodenum.[2] In mammals the duodenum may be the principal site for iron absorption.[3]

The duodenum precedes the jejunum and ileum and is the shortest part of the small intestine, where most chemical digestion takes place.[4]

In humans, the duodenum is a hollow jointed tube about 25–38 cm (10–15 inches) long connecting the stomach to the jejunum. It begins with the duodenal bulb and ends at the suspensory muscle of duodenum.[5]

The spleen

The spleen (from Greek σπλήνsplḗn[2]) is an organ found in virtually all vertebrates. Similar in structure to a large lymph node, it acts primarily as a blood filter.

It is possible to remove the spleen without jeopardizing life. The spleen plays important roles in regard to red blood cells (also referred to as erythrocytes) and the immune system.[3] It removes old red blood cells and holds a reserve of blood, which can be valuable in case of hemorrhagic shock, and also recycles iron. As a part of the mononuclear phagocyte system, it metabolizeshemoglobin removed from senescent erythrocytes. The globin portion of hemoglobin is degraded to its constitutive amino acids, and the heme portion is metabolized to bilirubin, which is removed in the liver.[4]

The spleen synthesizes antibodies in its white pulp and removes antibody-coated bacteria and antibody-coated blood cells by way of blood and lymph node circulation. A study published in 2009 using mice found that the spleen contains, in its reserve, half of the body’s monocytes within the red pulp.[5] These monocytes, upon moving to injured tissue (such as the heart), turn into dendritic cellsand macrophages while promoting tissue healing.[5][6][7] The spleen is a center of activity of the mononuclear phagocyte system and can be considered analogous to a large lymph node, as its absence causes a predisposition to certain infections.[8]

In humans, the spleen is brownish in color and is located in the left upper quadrant of the abdomen.[4][9]

The Psychochemical Response

Failure to Confirm an Hypothesis Based on Watson’s Psychochemical Typology
David L. Braff, M.D.,1 and Enoch Callaway, M.D.

Many of the theories behind so-called “Orthomolecular” treatment are so complex as to make testing them difficult, if not impossible. By contrast, a theory proposed by Watson in a series of papers (Watson, 1960, 1957; Watson and
Comrey, 1954; and Watson and Currier, 1960) and in a book entitled Nutrition and Your Mind —The Psychochemical Response (1972) is fairly explicit and seems to lend itself to empirical evaluation. On the basis of detailed
metabolic analyses of over 200 patients in the past 20 years, Watson divided his patients into two metabolic types. Watson originally made this distinction on the basis of patients’ responses to various Orthomolecular vitamin-mineral treatment regimens, but retrospectively found that these correlated with determinations of venous plasma pH, CO2 and H2CO3, total lipids, and fasting blood sugar. Watson labels these Psychochemical types as Type 1 with high pH (slow oxidizers) and Type II with low pH (fast oxidizers).

Watson’s theory states that Type I slow oxidizers are supposed to metabolize fats and ketogenic amino acids faster than carbohydrates and glucogenic amino acids. The vitamins and minerals that ameliorate a Type I patient’s symptoms (folic acid, niacin, thiamine) are generally cofactors at key steps in the Kreb’s and Embden-Meyeroff cycles that favor the utilization of the under-utilized carbohydrates and glucogenic amino acids. The pathophysiology of Type II is the reverse of Type I, and the main Orthomolecular treatments (pantothenic acid, choline, nicotinamide) act to increase utilization of fats and ketogenic amino acids. A more complete discussion of these issues is supplied by Watson in his book. Watson further claims that a food preference and reaction list (the
Psychochemical Profile) can generally determine if someone suffers from a Type I or Type II abnormality. The published form of the questionnaire (Watson, 1972, p. 74) was derived from the study of Watson’s series of
200 patients and is a simplified version of the research test, which has not been published.

Conventional metabolic experts raise the objection that venous pH, which Watson claims is the single most important
classificatory test, is an unreliable measure of metabolism. However, the failure to use the most up-to-date techniques does not preclude one from making a valuable clinical observation, and we decided to see if we could
repeat some of Watson’s observations. After we consulted with Dr. Watson, we decided first to determine the frequency of Type I and Type II cases in our clinical population, both by venous pH and by Watson’s published food preference list, prior to considering the possibility of clinical trials.

Of the 81 patients tested over the course of nine months, three were classified as Type I, with pH values exceeding 7.45 (roughly 4 percent), and eight were classified as Type II, with values below 7.35 (roughly 10 percent).
Table 1 summarizes these results.

Next, we examined the Psycho-chemical Profile results of those patients with Types I and II pH values, along with the 20 randomly selected patients with normal venous pH who were given the Profile. The most striking result is that none of the 31 patients, including 11 with clearly aberrant pH’s, was scored as either a Type I or II according to the Watson criteria.

Watson’s claim that his Psycho-chemical Profile distinguishes between Type I slow oxidizers with high pH’s and Type II fast oxidizers with low pH’s receives no support from our study. In fact, none of the patients with clearly abnormal pH’s (less than 7.35 or more than 7.45) was distinguishable from normal pH patients (pH between 7.35 and 7.45) on the basis of Watson’s criteria from his Profile.

cash register receipts

October 29, 2014

One thing you probably touch every day, over and over again, it’s cash register receipts.

Now researchers have found that those innocent-seeming pieces of paper contain high levels of bisphenol A, the same chemical recently banned from plastic water bottles because of the serious long-term health risks it poses.

According to a study published this week in the journal PLOS ONE, people’s blood levels ofBPA spiked after they touched cash receipts—particularly if they had lotion, sanitizer, or another skin care product on their hands.

“BPA has been proven to cause reproductive defects in fetuses, infants, children, and adults as well as cancer, metabolic, and immune problems in rodents,” said study author Frederick vom Saal, a professor of biology at the University of Missouri.

”Our research found BPA levels from receipts much higher than exposures from food packaging or plastic,” added vom Saal. “And BPA from thermal papers will be absorbed into your blood rapidly. At those levels, many diseases such as diabetes and disorders such as obesity increase as well.”

prevent type 2 diabetes

To help prevent type 2 diabetes and its
complications, people should:
• Achieve and maintain healthy body weight.
• Be physically active – at least 30 minutes of
regular, moderate-intensity activity on most
• Early diagnosis can be accomplished through
relatively inexpensive blood testing.
• Treatment of diabetes involves lowering blood
sugar and the levels of other known risk
factors that damage blood vessels.
• Tobacco cessation is also important to avoid

Diabetes is not a disease; it is a reaction of the body to survive

A body does not want to die. Nobody just likes to die, to give up, and kick the bucket. Physical discomfort is another story.

Diseases, or, that what we describe as disease are always the result of ‘something’. Colds can be sustained by coldness and rain. A broken leg can be caused by a fall. If you do not eat, you feel hungry.
You get appetite due to an empty stomach. If we eat food, the feeling will disappear. Then food was the right solution. This is not a disease, but a question of too little food.

Diabetes, a word often heard today, and it is used often, as it comes in handy. Was diabetes in earlier times also a term en vogue? Even small children are confronted with this diagnose; is this normal?
Diabetes and obesity often go together.

Our bodies however have to accept anything we eat. We use, for example, unhealthy spreads on our sandwich; spreads, only ‘one molecule different from plastic’.

If you have cirrhosis

It is vitally important that patients with liver disease maintain a balanced diet, one which ensures adequate calories, carbohydrates, fats and proteins. Such a diet will aid the liver in the regeneration of liver cells. Nutrition that supports this regeneration is a means of treatment of some liver disorders.

Patients with cirrhosis, for example, who are malnourished, require a diet rich in protein and providing 2,000 – 3,000 calories per day to help the liver re-build itself. However, some cirrhotic patients have protein intolerance. Too much protein will result in an increased amount of ammonia in the blood, while too little protein can reduce healing of the liver. Doctors must carefully prescribe a specific amount of protein that will not elevate the blood ammonia. Lactulose and neomycin are two drugs that help keep the ammonia down.

It is believed that the risk of gallbladder disorders can be reduced by avoiding high fat and cholesterol foods and preventing obesity. The gallbladder is a storage sac for the bile produced by the liver. During digestion, the gallbladder releases bile into the small intestine through the common bile duct. Most gallbladder problems are caused by gallstones and 80-90% of all gallstones are produced from excessive cholesterol which crystallizes and forms stones. By maintaining a well-balanced diet and avoiding high cholesterol intake, the incidence of gallstone formation may be lowered.

If you have cirrhosis, be careful to limit additional liver damage:

  • Don’t drink alcohol. Whether your cirrhosis was caused by chronic alcohol use or another disease, avoid alcohol. Drinking alcohol may cause further liver damage.
  • Eat a low-sodium diet. Excess salt can cause your body to retain fluids, worsening swelling in your abdomen and legs. Use herbs for seasoning your food, rather than salt. Choose prepared foods that are low in sodium.
  • Eat a healthy diet. People with cirrhosis can experience malnutrition. Combat this with a healthy plant-based diet that includes a variety of fruits and vegetables. Choose lean protein, such as legumes, poultry or fish. Avoid raw seafood.
  • Avoid infections. Cirrhosis make sit more difficult for you to fight off infections. Protect yourself by avoiding people who are sick and washing your hands frequently. Get vaccinated for hepatitis A and B, influenza, and pneumonia.
  • Use over-the-counter medications carefully. Cirrhosis makes it more difficult for your liver to process drugs. For this reason, ask your doctor before taking any medications, including nonprescription drugs. Avoid drugs such as aspirin and ibuprofen (Advil, Motrin IB). If you have liver damage, your doctor may recommend you avoid acetaminophen (Tylenol, others) or take it in low doses for pain relief.

People with cirrhosis may need more extra calories and protein. They may lose their appetite and experience nausea, vomiting, and severe weight loss. This can lead to shortage of the minerals calcium and magnesium (signs include muscle cramps, fatigue, weakness, nausea, and vomiting), or a shortage of zinc (signs include reduced ability to taste, changes in taste).

It can help to eat small, frequent meals (4 to 7 times a day), including an evening snack.

When the scarring from cirrhosis prevents blood from passing through the liver, pressure increases in the veins entering the liver. This is called portal hypertension. The body is forced to reroute the blood away from the liver and into the general blood circulation. This causes large blood vessels, called “varices,” to form.

Because the rerouted blood bypasses the liver, it contains high levels of amino acids, ammonia, and toxins that normally would have been handled by the liver. When these substances reach the brain, they can cause confusion and temporary loss of memory (a condition called “hepatic encephalopathy”).

Amino acids and ammonia come from protein in the diet. Some evidence shows that patients with cirrhosis do better when they get their protein from vegetables (such as beans, lentils, and tofu) and from dairy products (eggs, milk, yogurt) instead of from meats.

General recommendations for patients with severe liver disease include:

  • Eat large amounts of carbohydrate foods. Carbohydrates should be the major source of calories in this diet.
  • Eat a moderate intake of fat, as prescribed by the health care provider. The increased carbohydrates and fat help prevent protein breakdown in the liver.
  • Have about 1 gram of protein per kilogram of body weight. This means that a 154-pound (70-kilogram) man should eat 70 grams of protein per day. This does not include the protein from starchy foods and vegetables. A person with a badly damaged liver may need to eat less protein. Talk to your doctor about your protein needs.
  • Take vitamin supplements, especially B-complex vitamins.
  • Reduce the amount of salt you consume (typically less than 1500 milligrams per day) if you are retaining fluid.